Morphine paradoxically prolongs neuropathic pain in rats by amplifying spinal NLRP3 inflammasome activation
Peter M. Grace(The University of Texas MD Anderson Cancer Center), Linda R. Watkins(University of Colorado Boulder), Yingning Zhang(University of Colorado Boulder), Steven F. Maier(University of Colorado Boulder), Timothy Fabisiak(University of Colorado Boulder), Hang Yin(Cornell University), Debra Berkelhammer(University of Colorado Boulder), Keith A. Strand(Applied DNA Sciences (United States)), Serge Campeau(University of Colorado Boulder), Erika L. Galer(University of Colorado Boulder), Amanda Ellis(University of Colorado Boulder), Daniel J. Urban(National Institutes of Health), Kenner C. Rice(National Institute on Drug Abuse), Nathan D. Anderson(University of Colorado Boulder), Kejun Cheng(National Institute on Drug Abuse), Xiaohui Wang(Henan Academy of Sciences), Michael V. Baratta(University of Colorado Boulder), Lisa I. Greene(University of Colorado Anschutz Medical Campus), Bryan L. Roth(University of North Carolina at Chapel Hill)
Cited by 378
Related Papers
The promise and peril of chemical probes
|Nature Chemical Biology|2015|848
Evidence that opioids may have toll-like receptor 4 and MD-2 effects
|Brain Behavior and Immunity|2009|574
Morphine activates neuroinflammation in a manner parallel to endotoxin
|Proceedings of the National Academy of Sciences|2012|512
Strategies for Targeting Protein–Protein Interactions With Synthetic Agents
|Angewandte Chemie International Edition|2005|449