Training improves the oxidative phenotype of muscle during the transition from cardiac hypertrophy to heart failure without altering MyoD and myogenin
Francis Lopes Pacagnelli(Universidade do Oeste Paulista), Maeli Dal‐Pai‐Silva(Universidade Estadual Paulista (Unesp)), Dijon Henrique Salomé de Campos(Universidade de São Paulo), Fernanda Losi Alves de Almeida(Universidade Estadual Paulista (Unesp)), Andreo Fernando Aguiar(Universidade Norte do Paraná), Rodrigo Wagner Alves de Souza(Beth Israel Deaconess Medical Center), Robson Francisco Carvalho(Universidade Estadual Paulista (Unesp)), Eduardo Paulino Castan(Universidade Estadual Paulista (Unesp)), Fernanda Regina Carani(Universidade Estadual de Campinas (UNICAMP)), Antônio Carlos Cicogna(Universidade Estadual Paulista (Unesp))
Cited by 7
Related Papers
Aerobic Exercise Training Prevents Heart Failure-Induced Skeletal Muscle Atrophy by Anti-Catabolic, but Not Anabolic Actions
|PLoS ONE|2014|107
Acute Doxorubicin-Induced Cardiotoxicity is Associated with Matrix Metalloproteinase-2 Alterations in Rats
|Cellular Physiology and Biochemistry|2015|63
Heart failure increases atrogin-1 and MuRF1 gene expression in skeletal muscle with fiber type-specific atrophy
|Journal of Molecular Histology|2010|53
Regulation of cardiac microRNAs induced by aerobic exercise training during heart failure
|American Journal of Physiology-Heart and Circulatory Physiology|2015|47