Netrin-1 Interrupts Amyloid-β Amplification, Increases sAβPPα in vitro and in vivo, and Improves Cognition in a Mouse Model of Alzheimer’s Disease
Patricia Spilman, Dale E. Bredesen(Drug Discovery Laboratory (Norway)), Jesus Campagna(Foundation for Psychocultural Research), Varghese John(University of California, Los Angeles), Krystof Bankiewicz(University of California, San Francisco), Rammohan V. Rao(University of Washington), Olivia Gorostiza(Buck Institute for Research on Aging), Verónica Galván(University of Oklahoma Health Sciences Center), Karen S. Poksay(Buck Institute for Research on Aging), Véronique Corset(Centre de Recherche en Cancérologie de Lyon), Clare Peters‐Libeu(Buck Institute for Research on Aging), Junli Zhang(Huazhong Agricultural University), Patrick Mehlen(Université Claude Bernard Lyon 1), Jon Vincelette(BioMarin (United States)), Janine Beyer(Neurological Surgery), Andrew McGeehan(BioMarin (United States)), Melita Dvorak‐Ewell(BioMarin (United States))
Cited by 39
Related Papers
Coupling endoplasmic reticulum stress to the cell death program
|Cell Death and Differentiation|2004|1.2k
Functional gamma‐secretase inhibitors reduce beta‐amyloid peptide levels in brain
|Journal of Neurochemistry|2001|879
Ayurvedic medicinal plants for Alzheimer's disease: a review
|Alzheimer s Research & Therapy|2012|212
Optimization of the heme biosynthesis pathway for the production of 5-aminolevulinic acid in Escherichia coli
|Scientific Reports|2015|163