Mst1-mediated phosphorylation of Bcl-xL is required for myocardial reperfusion injury
Michinari Nakamura(Rutgers, The State University of New Jersey), Junichi Sadoshima(Rutgers, The State University of New Jersey)
Cited by 59
Related Papers
Autophagy in major human diseases
|The EMBO Journal|2021|1.6k
Brachial-Ankle Pulse Wave Velocity and the Risk Prediction of Cardiovascular Disease
|Hypertension|2017|538
Silent Information Regulator 2α, a Longevity Factor and Class III Histone Deacetylase, Is an Essential Endogenous Apoptosis Inhibitor in Cardiac Myocytes
|Circulation Research|2004|302
Nicotinamide Mononucleotide, an Intermediate of NAD+ Synthesis, Protects the Heart from Ischemia and Reperfusion
|PLoS ONE|2014|295
Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy
|Journal of Clinical Investigation|2003|277