Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis

Georg Auburger(Goethe University Frankfurt), Nadine Brehm(Goethe University Frankfurt)

Parkinson s Disease

January 1, 2016

10.1155/2016/4686185

Cited by 18

Related Papers

Potentiation of neurotoxicity in double-mutant mice with Pink1 ablation and A53T-SNCA overexpression

|Human Molecular Genetics|2014|70

CLPP Depletion Causes Diplotene Arrest; Underlying Testis Mitochondrial Dysfunction Occurs with Accumulation of Perrault Proteins ERAL1, PEO1, and HARS2

|Cells|2022|13

SerThr-PhosphoProteome of Brain from Aged PINK1-KO+A53T-SNCA Mice Reveals pT1928-MAP1B and pS3781-ANK2 Deficits, as Hub between Autophagy and Synapse Changes

|Preprints.org|2019|5

Multiomics approach identifies SERPINB1 as candidate biomarker for spinocerebellar ataxia type 2

|Scientific Reports|2025|3

Clpp-Deletion Causes Azoospermia, with Meiosis-I Delay and Insufficient Biosynthesis of Spermatid Factors, Due to Mitochondrial Dysfunction with Accumulation of Perrault Proteins ERAL1, PEO1, and HARS2

|Preprints.org|2022|2