A mouse model of a human congenital disorder of glycosylation caused by loss of PMM2
Barden Chan, Shengfang Jin(Agios Pharmaceuticals (United States)), Marion Dorsch(Biogen (United States)), Gromoslaw A. Smolen(Celsius Therapeutics (United States)), Zhi‐Zhong Lin(Xiamen University), Gavin Histen(Agios Pharmaceuticals (United States)), Yuanfeng Xia(WuXi AppTec (China)), Shinsan M. Su(Agios Pharmaceuticals (United States)), Lee Silverman(Agios Pharmaceuticals (United States)), Ryan Thompson(ID Genomics (United States)), Chris Singleton(Agios Pharmaceuticals (United States)), Michelle Clasquin(Agios Pharmaceuticals (United States)), Yong Cang(ShanghaiTech University), Chenming Lu(WuXi AppTec (China)), Yan Liu(First Hospital of China Medical University), Yue Chen(Agios Pharmaceuticals (United States)), Zhonghua Yan(Takeda (United States)), Josh Powe(Agios Pharmaceuticals (United States)), Hudson H. Freeze(Discovery Institute)
Cited by 47
Related Papers
Essentials of Glycobiology
|Unknown|1999|2.5k
Symbol Nomenclature for Graphical Representations of Glycans
|Glycobiology|2015|1.1k
Proinflammatory S100 Proteins Regulate the Accumulation of Myeloid-Derived Suppressor Cells
|The Journal of Immunology|2008|728
<i>Thermus aquaticus</i> gen. n. and sp. n., a Nonsporulating Extreme Thermophile
|Journal of Bacteriology|1969|688
Lysosomal Protease Pathways to Apoptosis
|Journal of Biological Chemistry|2001|658