Gliotoxin Suppresses NF-κB Activation by Selectively Inhibiting Linear Ubiquitin Chain Assembly Complex (LUBAC)
Hiroki Sakamoto(Shizuoka General Hospital), Tetsuo Nagano(The Open University of Japan), Takayoshi Okabe(The University of Tokyo), Toru Komatsu(The University of Tokyo), Simon Miller(High Energy Accelerator Research Organization), Takayoshi Kirisako(Osaka City University), Soichi Wakatsuki(Palo Alto University), Hirotatsu Kojima(The Open University of Japan), Kenjiro Hanaoka(The University of Tokyo), Tasuku Ueno(The University of Tokyo), Kazuhiro Iwaï(Kyoto University), Tadahiko Matsumoto(Kajima Corporation (Japan)), Shinichiro Egashira(The Open University of Japan), Yoshiteru Sasaki(Boston Children's Hospital), Takuya Terai(The University of Tokyo), Nae Saito(The Open University of Japan), Manabu Shimonishi(Kyoto University)
Cited by 82
Related Papers
Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
|Nature Genetics|2000|2k
PI3 Kinase Signals BCR-Dependent Mature B Cell Survival
|Cell|2009|651
PLEKHM1 Regulates Autophagosome-Lysosome Fusion through HOPS Complex and LC3/GABARAP Proteins
|Molecular Cell|2014|545
The cell-cycle regulator c-Myc is essential for the formation and maintenance of germinal centers
|Nature Immunology|2012|453