Ablation of cardiac myosin binding protein-C disrupts the super-relaxed state of myosin in murine cardiomyocytes
James W. McNamara(Royal Children's Hospital), Roger Cooke(Resources For The Future), Sean Lal(The University of Sydney), Kristina B. Kooiker(University of Washington), Amy Li(The University of Sydney), Sabine J. van Dijk(University of Arizona), Nicola J. Smith, Cristobal G. dos Remedios(The University of Sydney), Samantha P. Harris(University of Arizona), Robert M. Graham(Victor Chang Cardiac Research Institute)
Cited by 152
Related Papers
Allostery at G Protein-Coupled Receptor Homo- and Heteromers: Uncharted Pharmacological Landscapes
|Pharmacological Reviews|2010|255
A model of cardiac contraction based on novel measurements of tension development in human cardiomyocytes
|Journal of Molecular and Cellular Cardiology|2017|168
A conformational switch in HP1 releases auto-inhibition to drive heterochromatin assembly
|Nature|2013|162
Physiological Mitochondrial Fragmentation Is a Normal Cardiac Adaptation to Increased Energy Demand
|Circulation Research|2017|143