Deletion of the γ-secretase subunits <i>Aph1B/C</i> impairs memory and worsens the deficits of knock-in mice modeling the Alzheimer-like familial Danish dementia
Fabrizio Biundo(Albert Einstein College of Medicine), Luciano D'adamio(Rutgers, The State University of New Jersey), Dolores Del Prete(BioMed X Institute), Keita Ishiwari(Albert Einstein College of Medicine)
Cited by 17
Related Papers
Ryanodine Receptor Blockade Reduces Amyloid-β Load and Memory Impairments in Tg2576 Mouse Model of Alzheimer Disease
|Journal of Neuroscience|2012|238
A role for tau in learning, memory and synaptic plasticity
|Scientific Reports|2018|170
Localization and Processing of the Amyloid-β Protein Precursor in Mitochondria-Associated Membranes
|Journal of Alzheimer s Disease|2016|157
Ryanodine receptors: physiological function and deregulation in Alzheimer disease
|Molecular Neurodegeneration|2014|148
BRI2 Inhibits Amyloid β-Peptide Precursor Protein Processing by Interfering with the Docking of Secretases to the Substrate
|Journal of Neuroscience|2008|97