A novel splenic B1 regulatory cell subset suppresses allergic disease through phosphatidylinositol 3-kinase–Akt pathway activation
Takashi Matsushita(Kanazawa University), Manabu Fujimoto(Osaka Gakuin University), Minoru Hasegawa(University of Fukui), Yasuhito Hamaguchi(Kanazawa University), Masamichi Muramatsu(National Institute of Infectious Diseases), Tadahiro Kobayashi(Kanazawa University), Atsushi Hirao(Kanazawa University), Kazuhiko Takehara(Kanazawa University), Kazuhito Naka(Kanazawa University)
Cited by 68
Related Papers
Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)
|Cell|2000|1.6k
A Regulatory B Cell Subset with a Unique CD1dhiCD5+ Phenotype Controls T Cell-Dependent Inflammatory Responses
|Immunity|2008|1.2k
Regulatory B cells inhibit EAE initiation in mice while other B cells promote disease progression
|Journal of Clinical Investigation|2008|907
Idiopathic inflammatory myopathies
|Nature Reviews Disease Primers|2021|683
Connective Tissue Growth Factor Gene Expression in Tissue Sections From Localized Scleroderma, Keloid, and Other Fibrotic Skin Disorders
|Journal of Investigative Dermatology|1996|438