Chronic <i>Trypanosoma cruzi</i> infection potentiates adipose tissue macrophage polarization toward an anti-inflammatory M2 phenotype and contributes to diabetes progression in a diet-induced obesity model
María Eugenia Cabalén(Consejo Nacional de Investigaciones Científicas y Técnicas), Roxana Carolina Cano(Consejo Nacional de Investigaciones Científicas y Técnicas), Luisina I. Onofrio(Consejo Nacional de Investigaciones Científicas y Técnicas), Nicolás Eric Ponce(Consejo Nacional de Investigaciones Científicas y Técnicas), Marta C. Andrada(Universidad Católica de Córdoba), Liliana M. Sanmarco(Brigham and Women's Hospital), María F. Cabral(Consejo Nacional de Investigaciones Científicas y Técnicas), Susana Gea(Universidad Nacional de Córdoba), María Pilar Aoki(Consejo Nacional de Investigaciones Científicas y Técnicas)
Cited by 43
Related Papers
Pro-inflammatory monocyte profile in patients with major depressive disorder and suicide behaviour and how ketamine induces anti-inflammatory M2 macrophages by NMDAR and mTOR
|EBioMedicine|2019|173
Cytokines and cell adhesion receptors in the regulation of immunity to Trypanosoma cruzi
|Cytokine & Growth Factor Reviews|2007|98
IL-6 promotes M2 macrophage polarization by modulating purinergic signaling and regulates the lethal release of nitric oxide during Trypanosoma cruzi infection
|Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease|2017|87
Myeloid‐derived suppressor cells are key players in the resolution of inflammation during a model of acute infection
|European Journal of Immunology|2013|77