CYP3A5 mediates basal and acquired therapy resistance in different subtypes of pancreatic ductal adenocarcinoma

Elisa M. Noll(German Cancer Research Center), Christian Eisen(German Cancer Research Center), Albrecht Stenzinger(Heidelberg University), Elisa Espinet(German Cancer Research Center), Alexander Muckenhuber(Heidelberg University), Corinna Klein(Heidelberg Institute for Stem Cell Technology and Experimental Medicine), Vanessa Vogel(Heidelberg Institute for Stem Cell Technology and Experimental Medicine), Bernd Klaus(European Molecular Biology Laboratory), Wiebke Nadler(German Cancer Research Center), Christoph Rösli(Heidelberg Institute for Stem Cell Technology and Experimental Medicine), Christian Lutz(Heidelberg Pharma (Germany)), Michael Kulke(Heidelberg Pharma (Germany)), Jan Engelhardt(German Cancer Research Center), Franziska Maria Zickgraf(German Cancer Research Center), Octavio Espinosa(German Cancer Research Center), Matthias Schlesner(German Cancer Research Center), Xiaoqi Jiang(German Cancer Research Center), Annette Kopp‐Schneider(German Cancer Research Center), P. Neuhaus(Charité - Universitätsmedizin Berlin), Marcus Bahra(Charité - Universitätsmedizin Berlin), Bruno V. Sinn(Charité - Universitätsmedizin Berlin), Roland Eils(German Cancer Research Center), Nathalia A. Giese(Heidelberg University), Thilo Hackert(Heidelberg University), Oliver Strobel(Heidelberg University), Jens Werner(Heidelberg University), Markus W. Büchler(German Cancer Research Center), Wilko Weichert(German Cancer Research Center), Andreas Trumpp(German Cancer Research Center), Martin R. Sprick(German Cancer Research Center)
Nature Medicine
February 8, 2016
Cited by 227Open Access
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