Heart Rate and Extracellular Sodium and Potassium Modulation of Gap Junction Mediated Conduction in Guinea Pigs
Michael Entz(Virginia Tech), Steven Poelzing(Virginia Tech - Wake Forest University School of Biomedical Engineering & Sciences)
Cited by 32
Related Papers
Revealing the Concealed Nature of Long-QT Type 3 Syndrome
|Circulation Arrhythmia and Electrophysiology|2017|59
Intercellular Sodium Regulates Repolarization in Cardiac Tissue with Sodium Channel Gain of Function
|Biophysical Journal|2020|33
Attenuating loss of cardiac conduction during no-flow ischemia through changes in perfusate sodium and calcium
|American Journal of Physiology-Heart and Circulatory Physiology|2020|25
Inhibition of Na+ channels ameliorates arrhythmias in a drug-induced model of Andersen-Tawil syndrome
|Heart Rhythm|2012|21
Ephaptic Coupling Is a Mechanism of Conduction Reserve During Reduced Gap Junction Coupling
|Frontiers in Physiology|2022|20