The histone lysine methyltransferase KMT2D sustains a gene expression program that represses B cell lymphoma development
Ana Ortega-Molina(Memorial Sloan Kettering Cancer Center), Isaac W. Boss(Cornell University), Andrés Canela(National Cancer Institute), Heng Pan(Cornell University), Yanwen Jiang(Cornell University), Chunying Zhao(Memorial Sloan Kettering Cancer Center), Man Jiang(Memorial Sloan Kettering Cancer Center), Deqing Hu(Northwestern University), Xabier Agirre(Universidad de Navarra), Itamar Niesvizky(Cornell University), Ji‐Eun Lee(National Institute of Diabetes and Digestive and Kidney Diseases), Hua-Tang Chen(National Institutes of Health), Daisuke Ennishi(BC Cancer Agency), David W. Scott(BC Cancer Agency), Anja Mottok(BC Cancer Agency), Christoffer Hother(BC Cancer Agency), Shichong Liu(University of Pennsylvania), Xing-Jun Cao(University of Pennsylvania), Wayne Tam(Cornell University), Rita Shaknovich(Cornell University), Benjamin A. García(University of Pennsylvania), Randy D. Gascoyne(BC Cancer Agency), Kai Ge(National Institutes of Health), Ali Shilatifard(Northwestern University), Olivier Elemento(Cornell University), André Nussenzweig(National Cancer Institute), Ari Melnick(Cornell University), Hans‐Guido Wendel(Memorial Sloan Kettering Cancer Center)
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