The Dlk1-Gtl2 Locus Preserves LT-HSC Function by Inhibiting the PI3K-mTOR Pathway to Restrict Mitochondrial Metabolism
Pengxu Qian(Stowers Institute for Medical Research), Xi He(Stowers Institute for Medical Research), Ariel Paulson(Stowers Institute for Medical Research), Zhenrui Li(Stowers Institute for Medical Research), Tao Fang(Stowers Institute for Medical Research), John M. Perry(Stowers Institute for Medical Research), Fengli Guo(Stowers Institute for Medical Research), Meng Zhao(Stowers Institute for Medical Research), Lei Zhi(Tianjin Medical University), Aparna Venkatraman(Christian Medical College, Vellore), Jeffrey S. Haug(Stowers Institute for Medical Research), Tari Parmely(Stowers Institute for Medical Research), Hua Li(Stowers Institute for Medical Research), Rick T. Dobrowsky(University of Kansas), Wen‐Xing Ding(University of Kansas Medical Center), Tomohiro Kono(Tokyo University of Agriculture), Anne C. Ferguson‐Smith(University of Cambridge), Linheng Li(Stowers Institute for Medical Research)
Cited by 208Open Access