Endotoxin-Induced Mortality Is Related to Increased Oxidative Stress and End-Organ Dysfunction, Not Refractory Hypotension, in Heme Oxygenase-1–Deficient Mice
Philippe Wiesel(Harvard University), Mark A. Perrella(Brigham and Women's Hospital), Koji Maemura(Nagasaki University), Andrea Pellacani(Harvard University), Nicole DiFonzo(Brigham and Women's Hospital), Kathryn Marino(Brigham and Women's Hospital), Brian W. LeBlanc(Brigham and Women's Hospital), Claire M. Doerschuk(University of British Columbia), Shaw‐Fang Yet(Unknown), Pooja B. Marria(Brigham and Women's Hospital), Anand Patel(Harvard University), Mu-En Lee(Maine Farmland Trust), Chäng U. Sim(Harvard University)
Cited by 187
Related Papers
Promotion of vascular smooth muscle cell growth by homocysteine: a link to atherosclerosis.
|Proceedings of the National Academy of Sciences|1994|806
Tumor necrosis factor downregulates an endothelial nitric oxide synthase mRNA by shortening its half-life.
|Circulation Research|1993|776
Smooth muscle cell-derived carbon monoxide is a regulator of vascular cGMP.
|Proceedings of the National Academy of Sciences|1995|710
Natriuretic peptide system in human heart failure.
|Circulation|1993|461
Lipoxin A <sub>4</sub> Regulates Natural Killer Cell and Type 2 Innate Lymphoid Cell Activation in Asthma
|Science Translational Medicine|2013|441