Absence of regulated splicing of fibronectin EDA exon reduces atherosclerosis in mice
Vladimir R. Babaev(Vanderbilt University Medical Center), Andrés F. Muro(International Centre for Genetic Engineering and Biotechnology), Fabiola Porro(International Centre for Genetic Engineering and Biotechnology), Francisco E. Baralle(AREA Science Park), Sergio Fazio(Alzheimer's Association), MacRae F. Linton(Vanderbilt University)
Cited by 51
Related Papers
Current Perspectives on Statins
|Circulation|2000|1.2k
Treatment of diabetes and atherosclerosis by inhibiting fatty-acid-binding protein aP2
|Nature|2007|732
Characterization and Functional Implications of the RNA Binding Properties of Nuclear Factor TDP-43, a Novel Splicing Regulator ofCFTR Exon 9
|Journal of Biological Chemistry|2001|707
Lack of macrophage fatty-acid–binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosis
|Nature Medicine|2001|692
TDP‐43 regulates its mRNA levels through a negative feedback loop
|The EMBO Journal|2010|651