Molecular cytogenetic analysis of inv dup(15) chromosomes, using probes specific for the Prader-Willi/Angelman syndrome region: clinical implications.

Julie Leana‐Cox(University of Maryland, Baltimore), Stuart Schwartz(Research Triangle Park Foundation), Rina Plattner(University of Kentucky), Linda C. Sheppard, Fern Tsien, Catherine G. Palmer, Joleen L. Zackowski, Wendy L. Flejter, Lauren S. Jenkins

PubMed

May 1, 1994

Cited by 129

Related Papers

c-Abl is activated by growth factors and Src family kinases and has a role in the cellular response to PDGF

|Genes & Development|1999|426

Evaluation of mental retardation: Recommendations of a consensus conference

|American Journal of Medical Genetics|1997|403

Activation of Abl Tyrosine Kinases Promotes Invasion of Aggressive Breast Cancer Cells

|Cancer Research|2006|199

Imatinib Reverses Doxorubicin Resistance by Affecting Activation of STAT3-Dependent NF-κB and HSP27/p38/AKT Pathways and by Inhibiting ABCB1

|PLoS ONE|2013|133

Abl tyrosine kinases are required for infection by Shigella flexneri

|The EMBO Journal|2003|125