Olesoxime suppresses calpain activation and mutant huntingtin fragmentation in the BACHD rat
Laura E. Clemens(University of Tübingen), Huu Phuc Nguyen(University Hospitals of the Ruhr-University of Bochum), Tanja T. Wlodkowski(University of Tübingen), Andreas Weiss(Novartis (China)), Thierry Bordet(HiFiBiO Therapeutics (France)), Rebecca M. Pruss(OZ Biosciences (France)), Libo Yu-Taeger(University of Tübingen), Jonasz Jeremiasz Weber(University Hospitals of the Ruhr-University of Bochum), Magali Michaud(OZ Biosciences (France)), Schamim H. Eckert(Goethe University Frankfurt), Carsten Calaminus(Siemens (Germany)), Gunter P. Eckert(Goethe University Frankfurt), Janett Gaca(Goethe University Frankfurt), Janine Magg(Bernstein Center for Computational Neuroscience Tübingen), Erik Jansson(University of Tübingen), Olaf Rieß(University of Tübingen), Bernd J. Pichler(University of Tübingen)
Cited by 42
Related Papers
Genome-wide association study reveals genetic risk underlying Parkinson's disease
|Nature Genetics|2009|2k
14-3-3 proteins in the nervous system
|Nature reviews. Neuroscience|2003|625
Loss of function mutations in the gene encoding Omi/HtrA2 in Parkinson's disease
|Human Molecular Genetics|2005|566
<i>SNCA</i> variants are associated with increased risk for multiple system atrophy
|Annals of Neurology|2009|283
Increased susceptibility to sporadic Parkinson's disease by a certain combined ?-synuclein/apolipoprotein E genotype
|Annals of Neurology|1999|279