Cyclooxygenase-2 Inhibition Induces Apoptosis Signaling via Death Receptors and Mitochondria in Hepatocellular Carcinoma
Michael Kern, Martina Müller(University Hospital Regensburg), Kai Breuhahn(Heidelberg University), Peter H. Krammer(Slovak Academy of Sciences), Wolfgang Stremmel(University Hospital Heidelberg), Henning Schulze‐Bergkamen(Heidelberg University), Peter Schirmacher(Heidelberg University), Christian Trautwein(TUM Klinikum), Christoph Michalski(Heidelberg University), Andreas F. Koch(General Department of Preventive Medicine), Anke Haugg(National Institute of Pathology), Helmut Friess(TUM Klinikum), Binje Fleischer(Johannes Gutenberg University Mainz), Henning Walczak(CRUK Lung Cancer Centre of Excellence), Tobias Schilling(Heidelberg University)
Cited by 167
Related Papers
FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex
|Cell|1996|3.1k
Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor.
|The EMBO Journal|1995|2.2k
Death and anti-death: tumour resistance to apoptosis
|Nature reviews. Cancer|2002|2.1k
Monoclonal Antibody-Mediated Tumor Regression by Induction of Apoptosis
|Science|1989|1.7k
Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)
|Nature|1995|1.6k