Pancreatic cancer modeling using retrograde viral vector delivery and in vivo CRISPR/Cas9-mediated somatic genome editing

Shin‐Heng Chiou(Stanford University), Ian P. Winters(Stanford University), Jing Wang(Howard Hughes Medical Institute), Santiago Naranjo(Stanford University), Crissy Dudgeon(Rutgers, The State University of New Jersey), Fiona B. Tamburini(Stanford University), Jennifer J. Brady(Stanford University), Dian Yang(Stanford University), Barbara M. Grüner(Stanford University), Chen-Hua Chuang(Stanford University), Deborah R. Caswell(Stanford University), Hong Zeng(Stanford University), Pauline Chu(Stanford University), Grace Kim(University of California, San Francisco), Darren R. Carpizo(Rutgers, The State University of New Jersey), Seung K. Kim(Howard Hughes Medical Institute), Monte M. Winslow(Stanford University)
Genes & Development
July 15, 2015
Cited by 278Open Access
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Abstract

Pancreatic ductal adenocarcinoma (PDAC) is a genomically diverse, prevalent, and almost invariably fatal malignancy. Although conventional genetically engineered mouse models of human PDAC have been instrumental in understanding pancreatic cancer development, these models are much too labor-intensive, expensive, and slow to perform the extensive molecular analyses needed to adequately understand this disease. Here we demonstrate that retrograde pancreatic ductal injection of either adenoviral-Cre or lentiviral-Cre vectors allows titratable initiation of pancreatic neoplasias that progress into invasive and metastatic PDAC. To enable in vivo CRISPR/Cas9-mediated gene inactivation in the pancreas, we generated a Cre-regulated Cas9 allele and lentiviral vectors that express Cre and a single-guide RNA. CRISPR-mediated targeting of Lkb1 in combination with oncogenic Kras expression led to selection for inactivating genomic alterations, absence of Lkb1 protein, and rapid tumor growth that phenocopied Cre-mediated genetic deletion of Lkb1. This method will transform our ability to rapidly interrogate gene function during the development of this recalcitrant cancer.


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