Mitochondrial dysfunction associated with increased oxidative stress and α-synuclein accumulation in PARK2 iPSC-derived neurons and postmortem brain tissue
Yoichi Imaizumi(Keio University Hospital), Hideyuki Okano(Keio University), Masanobu Shouji(Takeda (Japan)), Shigeto Sato(Juntendo University), Nobutaka Hattori(Juntendo University Hospital), Manabu Funayama(Juntendo University), Arifumi Kosakai(Keio University), Tomoko Nihira(Kitasato University), Yohei Okada(Aichi Medical University), Hideki Hayakawa(Kitasato University), Atsushi Nakanishi(Takeda (Japan)), Noboru Mizushima(The University of Tokyo), Tomoyoshi Soga(Keio University), Makoto Suematsu(Central Institute for Experimental Animals), Hideki Mochizuki(Osaka Gakuin University), Akiyoshi Hirayama(Yamagata University), Masato Koike(Juntendo University), Kôzô Hayashi(Takeda (Japan)), Masashi Takanashi(Juntendo University), Takuya Yagi(Eisai (United States)), Daisuke Ito(Mie University), Takako Hishiki(Keio University), Masayuki Amagai(Keio University), Yasuo Uchiyama(Taisei (Japan)), Yoshikuni Mizuno(Juntendo University), Wado Akamatsu(Keio University Hospital), Norihiro Suzuki(Keio University Hospital), Manabu Ohyama(Kyorin University), Naoko Kuzumaki(Keio University Hospital), Tetsuro Kobayashi(Keio University)
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