Rescuing ΔF508 CFTR with trimethylangelicin, a dual-acting corrector and potentiator
James F. Collawn(University of Alabama at Birmingham), Sadis Matalon(University of Alabama at Birmingham), Lianwu Fu(Cystic Fibrosis Foundation), Rafał Bartoszewski(University of Wrocław)
American Journal of Physiology-Lung Cellular and Molecular Physiology
July 26, 2014
Cited by 7
Related Papers
Comprehensive review of antimicrobial activities of plant flavonoids
|Phytochemistry Reviews|2018|1.2k
miRNAs regulate the HIF switch during hypoxia: a novel therapeutic target
|Angiogenesis|2018|291
Somatic mosaicism for copy number variation in differentiated human tissues
|Human Mutation|2008|214
Primary endothelial cell–specific regulation of hypoxia‐inducible factor (HIF)‐1 and HIF‐2 and their target gene expression profiles during hypoxia
|The FASEB Journal|2019|212
A Synonymous Single Nucleotide Polymorphism in ΔF508 CFTR Alters the Secondary Structure of the mRNA and the Expression of the Mutant Protein
|Journal of Biological Chemistry|2010|200