Cannabidiol Attenuates Cisplatin-Induced Nephrotoxicity by Decreasing Oxidative/Nitrosative Stress, Inflammation, and Cell Death
Hao Pan(Zhejiang University), Pál Pacher(National Cancer Institute)
Cited by 248
Related Papers
Potent Metalloporphyrin Peroxynitrite Decomposition Catalyst Protects Against the Development of Doxorubicin-Induced Cardiac Dysfunction
|Circulation|2003|277
Cannabinoid 1 Receptor Promotes Cardiac Dysfunction, Oxidative Stress, Inflammation, and Fibrosis in Diabetic Cardiomyopathy
|Diabetes|2012|248
Cannabidiol attenuates high glucose-induced endothelial cell inflammatory response and barrier disruption
|American Journal of Physiology-Heart and Circulatory Physiology|2007|208
CB1 cannabinoid receptors promote oxidative stress and cell death in murine models of doxorubicin-induced cardiomyopathy and in human cardiomyocytes
|Cardiovascular Research|2009|198
CB<sub>1</sub> cannabinoid receptors promote oxidative/nitrosative stress, inflammation and cell death in a murine nephropathy model
|British Journal of Pharmacology|2010|153