Molecular mechanisms of nutlin-3 involve acetylation of p53, histones and heat shock proteins in acute myeloid leukemiaIngvild Haaland(University of Bergen), Bjørn Tore Gjertsen(Haukeland University Hospital)Molecular CancerMay 21, 201410.1186/1476-4598-13-116Cited by 36SaveCiteExport RISWatch citationsRelated PapersFlt3 Y591 duplication and Bcl-2 overexpression are detected in acute myeloid leukemia cells with high levels of phosphorylated wild-type p53|Blood|2006|82Overlapping features of therapy-related and de novo <i>NPM1</i>-mutated AML|Blood|2022|43Expression of TP53 Isoforms p53β or p53γ Enhances Chemosensitivity in TP53null Cell Lines|PLoS ONE|2013|30Tyrosine kinase inhibitors and interferon‐α increase tunneling nanotube (TNT) formation and cell adhesion in chronic myeloid leukemia (CML) cell lines|The FASEB Journal|2020|20Treatment-free remission after a second TKI discontinuation attempt in patients with Chronic Myeloid Leukemia re-treated with dasatinib – interim results from the DAstop2 trial|Leukemia|2024|13