p53 Activates the CD95 (APO-1/Fas) Gene in Response to DNA Damage by Anticancer Drugs
Martina Müller(University Hospital Regensburg), Peter H. Krammer(Slovak Academy of Sciences), Sylvia Wilder(German Cancer Research Center), David Israeli(Inserm), Moshe Oren(Weizmann Institute of Science), Wolfgang Stremmel(University Hospital Heidelberg), Scott L. Friedman(Icahn School of Medicine at Mount Sinai), Katrin Lehlbach(German Cancer Research Center), Peter R. Galle(Johannes Gutenberg University Mainz), Min Li‐Weber(German Cancer Research Center)
Cited by 897
Related Papers
FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex
|Cell|1996|3.1k
Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor.
|The EMBO Journal|1995|2.2k
Molecular Regulation of Hepatic Fibrosis, an Integrated Cellular Response to Tissue Injury
|Journal of Biological Chemistry|2000|2.1k
Death and anti-death: tumour resistance to apoptosis
|Nature reviews. Cancer|2002|2.1k
Simplified criteria for the diagnosis of autoimmune hepatitis†
|Hepatology|2008|1.9k