Fasudil-induced hypoxia-inducible factor-1α degradation disrupts a hypoxia-driven vascular endothelial growth factor autocrine mechanism in endothelial cells

Keiko Takata(Yamagata University), Hirohisa Kurachi(Osaka Women's and Children's Hospital), Tsuyoshi Ohta(Yamagata University), Seiji Tsutsumi(Yamagata University), Kazuhiro Takahashi, Jun Kawagoe, Limei Yin(Yamagata University), Kae Hashimoto(The University of Osaka), Ken‐ichirou Morishige(Gifu University), Toshifumi Takahashi(Fukushima Medical University)
Molecular Cancer Therapeutics
June 1, 2008
10.1158/1535-7163.mct-07-0428
Cited by 46

Related Papers

Estrogen Induces the Akt-dependent Activation of Endothelial Nitric-oxide Synthase in Vascular Endothelial Cells
|Journal of Biological Chemistry|2001|387
Inhibition of BAD phosphorylation either at serine 112 via extracellular signal-regulated protein kinase cascade or at serine 136 via Akt cascade sensitizes human ovarian cancer cells to cisplatin.
|PubMed|2000|249
Exosomes Promote Ovarian Cancer Cell Invasion through Transfer of CD44 to Peritoneal Mesothelial Cells
|Molecular Cancer Research|2016|232
A Physiological Role of Epidermal Growth Factor in Male Reproductive Function
|Science|1986|222
Inhibition of NFκB Increases the Efficacy of Cisplatin in in Vitro and in Vivo Ovarian Cancer Models
|Journal of Biological Chemistry|2004|212