A heteroplasmic, not homoplasmic, mitochondrial DNA mutation promotes tumorigenesis via alteration in reactive oxygen species generation and apoptosis
J. S. Park(The University of Texas Health Science Center at San Antonio), Yidong Bai(The University of Texas Health Science Center at San Antonio)
Cited by 242
Related Papers
Mitochondrial respiratory complex I dysfunction promotes tumorigenesis through ROS alteration and AKT activation
|Human Molecular Genetics|2011|153