Cardiac-Specific IGF-1 Expression Attenuates Dilated Cardiomyopathy in Tropomodulin-Overexpressing Transgenic Mice
Sara Welch(New York Medical College), Mark A. Sussman(California Institute for Regenerative Medicine)
Cited by 147
Related Papers
Myocardial AKT: The Omnipresent Nexus
|Physiological Reviews|2011|222
Atrial natriuretic peptide promotes cardiomyocyte survival by cGMP-dependent nuclear accumulation of zyxin and Akt
|Journal of Clinical Investigation|2005|159
Pim-1 preserves mitochondrial morphology by inhibiting dynamin-related protein 1 translocation
|Proceedings of the National Academy of Sciences|2013|132
Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes
|Circulation Research|2015|128
Fibronectin Is Essential for Reparative Cardiac Progenitor Cell Response After Myocardial Infarction
|Circulation Research|2013|119