Ablation of oncogenic ALK is a viable therapeutic approach for anaplastic large-cell lymphomas

Roberto Piva(New York University), Giorgio Inghirami(New York University)

Blood

September 28, 2005

10.1182/blood-2005-05-2125

Cited by 128

Related Papers

NPM-ALK transgenic mice spontaneously develop T-cell lymphomas and plasma cell tumors

|Blood|2003|247

Functional validation of the anaplastic lymphoma kinase signature identifies CEBPB and Bcl2A1 as critical target genes

|Journal of Clinical Investigation|2006|164

15-Deoxy-Δ12,14-prostaglandin J2 induces apoptosis in human malignant B cells: an effect associated with inhibition of NF-κB activity and down-regulation of antiapoptotic proteins

|Blood|2004|94

STAT3-mediated activation of microRNA cluster 17 92 promotes proliferation and survival of ALK-positive anaplastic large cell lymphoma

|Haematologica|2013|53

The enzymatic activity of 5-aminoimidazole-4-carboxamide ribonucleotide formyltransferase/IMP cyclohydrolase is enhanced by NPM-ALK: new insights in ALK-mediated pathogenesis and the treatment of ALCL

|Blood|2008|52