ERK and p38 mediate high-glucose-induced hypertrophy and TGF-β expression in renal tubular cells

Hisayo Fujita(Keio University Hospital), Midori Awazu(Tokyo Metropolitan Ohtsuka Hospital), Mariko Hida(Keio University), Sayu Omori(Keio University Hospital), Kenji Ishikura(Kitasato University)

American Journal of Physiology-Renal Physiology

January 1, 2004

10.1152/ajprenal.00351.2002

Cited by 126

Related Papers

Rituximab for childhood-onset, complicated, frequently relapsing nephrotic syndrome or steroid-dependent nephrotic syndrome: a multicentre, double-blind, randomised, placebo-controlled trial

|The Lancet|2014|450

IPNA clinical practice recommendations for the diagnosis and management of children with steroid-sensitive nephrotic syndrome

|Pediatric Nephrology|2022|278

Creatinine-based equation to estimate the glomerular filtration rate in Japanese children and adolescents with chronic kidney disease

|Clinical and Experimental Nephrology|2013|267

Epidemiology of pediatric chronic kidney disease/kidney failure: learning from registries and cohort studies

|Pediatric Nephrology|2021|149

A multicenter randomized trial indicates initial prednisolone treatment for childhood nephrotic syndrome for two months is not inferior to six-month treatment

|Kidney International|2014|135