Mitochondrial Electron Transport Complex I Is a Potential Source of Oxygen Free Radicals in the Failing Myocardium
Tomomi Ide(Juntendo University), Akira Takeshita(Toranomon Hospital), Kenichi Arimura(Kyushu University), Hideo Utsumi(Shizuoka University), Kôji Uchida(The University of Tokyo), Hiroyuki Tsutsui(Juntendo University), Dongchon Kang(Kyushu University), Shintaro Kinugawa(Kyushu University), Kensuke Egashira(Kyushu University), Nobutaka Hattori(Juntendo University Hospital)
Cited by 700
Related Papers
Empagliflozin in Heart Failure with a Preserved Ejection Fraction
|New England Journal of Medicine|2021|4.6k
Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
|Nature Genetics|2000|2k
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy
|The Journal of Cell Biology|2010|1.9k