C3G forms complexes with Bcr-Abl and p38α MAPK at the focal adhesions in chronic myeloid leukemia cells: implication in the regulation of leukemic cell adhesion
Vera Maia(Universidad de Salamanca), Carmen Guerrero(Universidad de Salamanca)
Cited by 28
Related Papers
C3G contributes to platelet activation and aggregation by regulating major signaling pathways
|Signal Transduction and Targeted Therapy|2020|42
C3G, through its GEF activity, induces megakaryocytic differentiation and proplatelet formation
|Cell Communication and Signaling|2018|22