Reduced Adiposity and Liver Steatosis by Stearoyl-CoA Desaturase Deficiency Are Independent of Peroxisome Proliferator-activated Receptor-α
Makoto Miyazaki(Chiba Cancer Center), James M. Ntambi(University of Wisconsin–Madison), Weng Chi Man, Agnieszka Dobrzyń(Instytut Biologii Doświadczalnej im. Marcelego Nenckiego), Frank J. Gonzalez(National Cancer Institute), Jeffrey M. Peters(Pennsylvania State University), Harini Sampath(Rutgers, The State University of New Jersey), Kiki Chu(University of Wisconsin–Madison), Seong‐Ho Lee(University of Maryland, College Park)
Cited by 120
Related Papers
Loss of stearoyl–CoA desaturase-1 function protects mice against adiposity
|Proceedings of the National Academy of Sciences|2002|1.1k
Role for Stearoyl-CoA Desaturase-1 in Leptin-Mediated Weight Loss
|Science|2002|833
Differential regulation of bile acid homeostasis by the farnesoid X receptor in liver and intestine
|Journal of Lipid Research|2007|595
The Biosynthesis of Hepatic Cholesterol Esters and Triglycerides Is Impaired in Mice with a Disruption of the Gene for Stearoyl-CoA Desaturase 1
|Journal of Biological Chemistry|2000|453