HDAC2 regulates atypical antipsychotic responses through the modulation of mGlu2 promoter activity
Mitsumasa Kurita(Icahn School of Medicine at Mount Sinai), Terrell Holloway(Icahn School of Medicine at Mount Sinai), Aintzane García‐Bea(University of the Basque Country), Alexey Kozlenkov(Icahn School of Medicine at Mount Sinai), Allyson K. Friedman(Icahn School of Medicine at Mount Sinai), José L. Moreno(Icahn School of Medicine at Mount Sinai), Mitra Heshmati(Icahn School of Medicine at Mount Sinai), Sam A. Golden(Icahn School of Medicine at Mount Sinai), Pamela Kennedy(Icahn School of Medicine at Mount Sinai), Nagahide Takahashi(Icahn School of Medicine at Mount Sinai), David Dietz(Icahn School of Medicine at Mount Sinai), Giuseppe Mocci(Icahn School of Medicine at Mount Sinai), Ane M. Gabilondo(Centro de Investigación Biomédica en Red de Salud Mental), James B. Hanks(Icahn School of Medicine at Mount Sinai), Adrienne Umali(Icahn School of Medicine at Mount Sinai), Luís F. Callado(Centro de Investigación Biomédica en Red de Salud Mental), Amelia L. Gallitano(University of Arizona), Rachael L. Neve(Institute of Cognitive and Brain Sciences), Li Shen(Icahn School of Medicine at Mount Sinai), Joseph D. Buxbaum(Icahn School of Medicine at Mount Sinai), Ming‐Hu Han(Icahn School of Medicine at Mount Sinai), Eric J. Nestler(Allen Institute for Brain Science), J. Javier Meana(Centro de Investigación Biomédica en Red de Salud Mental), Scott J. Russo(Allen Institute for Brain Science), Javier González‐Maeso(Icahn School of Medicine at Mount Sinai)
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