Perinatal abrogation of Cdk5 expression in brain results in neuronal migration defects
Motoyuki Hirasawa(ID Genomics (United States)), Ashok B. Kulkarni(National Institutes of Health), Katsuhiko Mikoshiba(ShanghaiTech University), Toshio Ohshima(Waseda University), Veeranna(National Institute of Dental and Craniofacial Research), Roscoe O. Brady(Johns Hopkins University), Glenn Longenecker(National Institutes of Health), Yasuyuki Honjo(National Institutes of Health), Harish C. Pant(All India Institute of Medical Sciences Raipur), Satoru Takahashi(National Institutes of Health)
Cited by 47
Related Papers
A variant of yellow fluorescent protein with fast and efficient maturation for cell-biological applications
|Nature Biotechnology|2002|2.8k
‘Ischemic tolerance’ phenomenon found in the brain
|Brain Research|1990|1.1k
Primary structure and functional expression of the inositol 1,4,5-trisphosphate-binding protein P400
|Nature|1989|1.1k
2APB, 2-Aminoethoxydiphenyl Borate, a Membrane-Penetrable Modulator of Ins(1,4,5)P3-Induced Ca2+ Release
|The Journal of Biochemistry|1997|917
Targeted disruption of the cyclin-dependent kinase 5 gene results in abnormal corticogenesis, neuronal pathology and perinatal death.
|Proceedings of the National Academy of Sciences|1996|900