Excessive reactive oxygen species are therapeutic targets for intervertebral disc degeneration
Satoshi Suzuki(Keio University), Morio Matsumoto(Keio University Hospital), Masaya Nakamura(Keio University), Keisuke Horiuchi(National Defense Medical College), Nobuyuki Fujita(Fujita Health University), Keiyo Takubo(Keio University), Kota Watanabe(Keio University Hospital), Naobumi Hosogane(National Defense Medical College), Takeshi Miyamoto(Kumamoto Health Science University), Yoshiaki Toyama(Keio University), Ken Ishii(International University of Health and Welfare)
Cited by 245
Related Papers
DC-STAMP is essential for cell–cell fusion in osteoclasts and foreign body giant cells
|The Journal of Experimental Medicine|2005|899
Bidirectional ephrinB2-EphB4 signaling controls bone homeostasis
|Cell Metabolism|2006|771
Commitment and Differentiation of Osteoclast Precursor Cells by the Sequential Expression of C-Fms and Receptor Activator of Nuclear Factor κb (Rank) Receptors
|The Journal of Experimental Medicine|1999|694
v-ATPase V0 subunit d2–deficient mice exhibit impaired osteoclast fusion and increased bone formation
|Nature Medicine|2006|542
Osteoclast Differentiation Factor (ODF) Induces Osteoclast-like Cell Formation in Human Peripheral Blood Mononuclear Cell Cultures
|Biochemical and Biophysical Research Communications|1998|344