AICAR inhibits PPARγ during monocyte differentiation to attenuate inflammatory responses to atherogenic lipids

Dmitry Namgaladze(Goethe University Frankfurt), Bernhard Brüne(Goethe University Frankfurt)

Cardiovascular Research

March 25, 2013

10.1093/cvr/cvt073

Cited by 28

Related Papers

Mitochondrial composition and function under the control of hypoxia

|Redox Biology|2017|635

Cleavage of sphingosine kinase 2 by caspase-1 provokes its release from apoptotic cells

|Blood|2010|87

Functional Integrity of Nuclear Factor κB, Phosphatidylinositol 3′-Kinase, and Mitogen-Activated Protein Kinase Signaling Allows Tumor Necrosis Factor α-Evoked Bcl-2 Expression to Provoke Internal Ribosome Entry Site-Dependent Translation of Hypoxia-Inducible Factor 1α

|Cancer Research|2004|62

TMEM126B deficiency reduces mitochondrial SDH oxidation by LPS, attenuating HIF-1α stabilization and IL-1β expression

|Redox Biology|2018|61

Interplay of Nrf2 and BACH1 in inducing ferroportin expression and enhancing resistance of human macrophages towards ferroptosis

|Cell Death Discovery|2022|61