A Gradient of Myosin Regulatory Light-chain Phosphorylation across the Ventricular Wall Supports Cardiac Torsion

Julien S. Davis(National Institutes of Health), Nava Epstein(Sheba Medical Center), Shakiba Hassanzadeh(National Institutes of Health), Anthony H. Aletras(National Institutes of Health), Steve Winitsky(National Institutes of Health), Han Wen(University Medical Center New Orleans)

Cold Spring Harbor Symposia on Quantitative Biology

January 1, 2002

10.1101/sqb.2002.67.345

Cited by 21

Related Papers

Targeted integration of adeno‐associated virus (AAV) into human chromosome 19.

|The EMBO Journal|1991|774

Long-term results of dual-chamber (DDD) pacing in obstructive hypertrophic cardiomyopathy. Evidence for progressive symptomatic and hemodynamic improvement and reduction of left ventricular hypertrophy.

|Circulation|1994|383

Skeletal muscle expression and abnormal function of beta-myosin in hypertrophic cardiomyopathy.

|Journal of Clinical Investigation|1993|242

Differences in clinical expression of hypertrophic cardiomyopathy associated with two distinct mutations in the beta-myosin heavy chain gene. A 908Leu----Val mutation and a 403Arg----Gln mutation.

|Circulation|1992|230

Genotype-phenotype correlations in hypertrophic cardiomyopathy. Insights provided by comparisons of kindreds with distinct and identical beta-myosin heavy chain gene mutations.

|Circulation|1994|226