Cellular recombination pathways and viral terminal repeat hairpin structures are sufficient for adeno-associated virus integration in vivo and in vitro

Chao Yang(Soochow University), R. Jude Samulski(University of North Carolina at Chapel Hill), Xiangwei Xiao(University of Pittsburgh), A. Gregory Matera(University of North Carolina at Chapel Hill), Xiaodong Zhu(University of Pittsburgh), D C Ansardi(University of North Carolina at Chapel Hill), Manfred Frey(University of North Carolina at Chapel Hill), Nava Epstein(University of Wisconsin–Madison)
Journal of Virology
December 1, 1997
10.1128/jvi.71.12.9231-9247.1997
Cited by 178

Related Papers

Targeted integration of adeno‐associated virus (AAV) into human chromosome 19.
|The EMBO Journal|1991|774
Long-term results of dual-chamber (DDD) pacing in obstructive hypertrophic cardiomyopathy. Evidence for progressive symptomatic and hemodynamic improvement and reduction of left ventricular hypertrophy.
|Circulation|1994|383
M2 macrophages promote beta-cell proliferation by up-regulation of SMAD7
|Proceedings of the National Academy of Sciences|2014|318
Skeletal muscle expression and abnormal function of beta-myosin in hypertrophic cardiomyopathy.
|Journal of Clinical Investigation|1993|242
Differences in clinical expression of hypertrophic cardiomyopathy associated with two distinct mutations in the beta-myosin heavy chain gene. A 908Leu----Val mutation and a 403Arg----Gln mutation.
|Circulation|1992|230