Consequences of cardiac myocyte-specific ablation of K<sub>ATP</sub>channels in transgenic mice expressing dominant negative Kir6 subunits
Xiaoyong Tong(Chongqing University), William A. Coetzee(New York University), Cindy Yu(Jacksonville College), GongXin Liu, Lisa M. Porter, Glenn I. Fishman(New York University), Piyali Dhar-Chowdhury, Shekhar Srivastava(NYU Langone Health), Ramesh S. Iyer, Hidetada Yoshida, David Pountney, Gregory E. Morley(Scripps Research Institute), Michael Artman(New York University), David E. Gutstein(Scripps Research Institute)
American Journal of Physiology-Heart and Circulatory Physiology
February 25, 2006
Cited by 70
Related Papers
CRISPR-Cas9 In Vivo Gene Editing for Transthyretin Amyloidosis
|New England Journal of Medicine|2021|1.7k
Renin–Angiotensin–Aldosterone System Inhibitors and Risk of Covid-19
|New England Journal of Medicine|2020|1.1k
ST-Segment Elevation in Patients with Covid-19 — A Case Series
|New England Journal of Medicine|2020|858
Conduction Slowing and Sudden Arrhythmic Death in Mice With Cardiac-Restricted Inactivation of Connexin43
|Circulation Research|2001|686
Sudden Cardiac Death Prediction and Prevention
|Circulation|2010|608