miR-150 Down-Regulation Contributes to the Constitutive Type I Collagen Overexpression in Scleroderma Dermal Fibroblasts via the Induction of Integrin β3
Noritoshi Honda(Kumamoto University), Hironobu Ihn(Kumamoto University), Yoshinobu Okamoto(Kanazawa University), Tomomi Kira-Etoh(Kumamoto University), Katsunari Makino(Kumamoto University), Minoru Hasegawa(University of Fukui), Satoshi Fukushima(Niigata Cancer Center Hospital), Takamitsu Makino(Kumamoto University), Masatoshi Jinnin(Wakayama Medical University), Ikko Kajihara(Kumamoto University), Yuji Inoue(Kumamoto University), Manabu Fujimoto(Osaka Gakuin University)
Cited by 132
Related Papers
A Regulatory B Cell Subset with a Unique CD1dhiCD5+ Phenotype Controls T Cell-Dependent Inflammatory Responses
|Immunity|2008|1.2k
Regulatory B cells inhibit EAE initiation in mice while other B cells promote disease progression
|Journal of Clinical Investigation|2008|907
Idiopathic inflammatory myopathies
|Nature Reviews Disease Primers|2021|683
Connective Tissue Growth Factor Gene Expression in Tissue Sections From Localized Scleroderma, Keloid, and Other Fibrotic Skin Disorders
|Journal of Investigative Dermatology|1996|438