Transforming growth factor β1 and laminin-111 cooperate in the induction of interleukin-16 expression in synovial fibroblasts from patients with rheumatoid arthritis

Katrin Warstat(Center for Neuroscience and Regenerative Medicine), Wilhelm K. Aicher(STZ eyetrial), Steffen Gay(Centre Hospitalier Universitaire de Tours), Maximilian Rudert, Gerd Klein(University Children's Hospital Tübingen), Mike Essl, Terry J. Smith(University of Michigan), William W. Cruikshank(Boston University), M. Hoberg, Brigitte Angres(Natural and Medical Sciences Institute), Shanli Tsui(University of California, Los Angeles), Thomas Pap(University Hospital Münster)
Annals of the Rheumatic Diseases
March 12, 2009
10.1136/ard.2008.091116
Cited by 20

Related Papers

Graves’ Disease
|New England Journal of Medicine|2016|1.2k
Narcolepsy — clinical spectrum, aetiopathophysiology, diagnosis and treatment
|Nature Reviews Neurology|2019|634
Children with early-onset inflammatory bowel disease (IBD): Analysis of a pediatric IBD consortium registry
|The Journal of Pediatrics|2005|571
Toll-Like Receptor Engagement Enhances the Immunosuppressive Properties of Human Bone Marrow-Derived Mesenchymal Stem Cells by Inducing Indoleamine-2,3-dioxygenase-1 via Interferon-β and Protein Kinase R  
|Stem Cells|2009|303
Immunoglobulin Activation of T Cell Chemoattractant Expression in Fibroblasts from Patients with Graves’ Disease Is Mediated Through the Insulin-Like Growth Factor I Receptor Pathway
|The Journal of Immunology|2003|294