GLP-1 Mediates Antiapoptotic Effect by Phosphorylating Bad through a β-Arrestin 1-mediated ERK1/2 Activation in Pancreatic β-CellsJulie Quoyer(Institut de Génomique Fonctionnelle), Stéphane Dalle(Mount Sinai Hospital)Journal of Biological ChemistryNovember 14, 200910.1074/jbc.m109.067207Cited by 182SaveCiteExport RISWatch citationsRelated PapersDegradation of cAMP-Responsive Element–Binding Protein by the Ubiquitin-Proteasome Pathway Contributes to Glucotoxicity in β-Cells and Human Pancreatic Islets|Diabetes|2009|57