High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines
Ami Ketley(University of Nottingham), J. David Brook(University of Nottingham), I. Udosen(University of Nottingham), Wei Zheng(Liaoning Cancer Hospital & Institute), Noel Southall(National Institutes of Health), Thelma Robinson(University of Nottingham), Christopher P. Austin(Unknown), Christopher J. Hayes(University of Nottingham), Soraya Chaouch(Centre National de la Recherche Scientifique), Javier T Granados-Riveron(University of Nottingham), Denis Furling(Cytoskeleton (United States)), Sukrat Arya(University of Nottingham), Xin Li(Hubei University of Medicine), Ben Haworth(Molecular Devices (United Kingdom)), Glenn E. Morris(Robert Jones and Agnes Hunt Orthopaedic Hospital), Catherine Z. Chen(National Institutes of Health), Paul Shinn(National Institutes of Health), Ian Holt(Ikerbasque)
Cited by 70
Related Papers
The promise and peril of chemical probes
|Nature Chemical Biology|2015|848
Targeted integration of adeno‐associated virus (AAV) into human chromosome 19.
|The EMBO Journal|1991|774
Identification of the gene responsible for Best macular dystrophy
|Nature Genetics|1998|685