Human mitochondrial oxidative capacity is acutely impaired after burn trauma
Melanie Cree‐Green(University of Colorado Boulder), Robert R. Wolfe(University of Arkansas for Medical Sciences), Ronald P. Mlcak(Shriners Hospitals for Children - Galveston), Carlos A. Angel, Asle Aarsland, Justin M. Green(The University of Texas Medical Branch at Galveston), David N. Herndon(Joseph M. Still Research Foundation), Ting Qian, Ricki Y. Fram(Shriners Hospitals for Children - Galveston)
Cited by 44
Related Papers
Genomic responses in mouse models poorly mimic human inflammatory diseases
|Proceedings of the National Academy of Sciences|2013|3k
Fructose and sugar: A major mediator of non-alcoholic fatty liver disease
|Journal of Hepatology|2018|938
The leading causes of death after burn injury in a single pediatric burn center
|Critical Care|2009|376
Ingestion of Casein and Whey Proteins Result in Muscle Anabolism after Resistance Exercise
|Medicine & Science in Sports & Exercise|2004|349
Stimulation of net muscle protein synthesis by whey protein ingestion before and after exercise
|American Journal of Physiology-Endocrinology and Metabolism|2006|305