Interleukin-18 (Interferon-γ–inducing Factor) Is Produced by Osteoblasts and Acts Via Granulocyte/Macrophage Colony-stimulating Factor and Not Via Interferon-γ to Inhibit Osteoclast Formation
Nobuyuki Udagawa(Matsumoto Dental University), Matthew T. Gillespie(Hudson Institute of Medical Research), Haruki Okamura(Mukogawa Women's University), Nicole J. Horwood(University of East Anglia), Alan Mackay(Institute of Cancer Research), Jan Elliott(St George's Hospital), Masashi Kurimoto(Tokuyama (Japan)), Timothy Chambers(St George's Hospital), Jane G. Owens(St George's Hospital), T John Martin(St George's Hospital)
Cited by 376
Related Papers
Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL
|Proceedings of the National Academy of Sciences|1998|4.3k
Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand Families
|Endocrine Reviews|1999|2.3k
IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis
|Journal of Clinical Investigation|1999|1.7k
Tumor Necrosis Factor α Stimulates Osteoclast Differentiation by a Mechanism Independent of the Odf/Rankl–Rank Interaction
|The Journal of Experimental Medicine|2000|1.3k