Primary Endothelial Damage Is the Mechanism of Cardiotoxicity of Tubulin-Binding Drugs
Igor Mikaelian(Roche (Switzerland)), Laura Suter‐Dick(FHNW University of Applied Sciences and Arts)
Cited by 63
Related Papers
Pro-fibrotic compounds induce stellate cell activation, ECM-remodelling and Nrf2 activation in a human 3D-multicellular model of liver fibrosis
|PLoS ONE|2017|93
Nrf2 protects stellate cells from Smad-dependent cell activation
|PLoS ONE|2018|59
Methotrexate-Induced Liver Injury Is Associated with Oxidative Stress, Impaired Mitochondrial Respiration, and Endoplasmic Reticulum Stress In Vitro
|International Journal of Molecular Sciences|2022|55
In Vitro Hepatic Metabolism Input Parameters Support Toxicokinetic Simulations for the Formation of Methoxy Propionic Acid From β‐Isomer Propylene Glycol Methyl Ether
|Pharmacology Research & Perspectives|2024|2