Endothelial cell responses to atheroprone flow are driven by two separate flow components: low time-average shear stress and fluid flow reversal
Daniel E. Conway(The Ohio State University), Larry V. McIntire(Freeman Health System), Marcie R. Williams(Georgia Institute of Technology), Suzanne G. Eskin(University of Illinois Chicago)
American Journal of Physiology-Heart and Circulatory Physiology
November 13, 2009
Cited by 111
Related Papers
ADAMTS-13 rapidly cleaves newly secreted ultralarge von Willebrand factor multimers on the endothelial surface under flowing conditions
|Blood|2002|853
Host response to tissue engineered devices
|Advanced Drug Delivery Reviews|1998|584
Platelet Glycoprotein Ibα Is a Counterreceptor for the Leukocyte Integrin Mac-1 (Cd11b/Cd18)
|The Journal of Experimental Medicine|2000|582
Fluid Shear Stress on Endothelial Cells Modulates Mechanical Tension across VE-Cadherin and PECAM-1
|Current Biology|2013|508