Heterodimeric JAK–STAT activation as a mechanism of persistence to JAK2 inhibitor therapy

Priya Koppikar(Memorial Sloan Kettering Cancer Center), Neha Bhagwat(Memorial Sloan Kettering Cancer Center), Outi Kilpivaara(Memorial Sloan Kettering Cancer Center), Taghi Manshouri(The University of Texas MD Anderson Cancer Center), Mazhar Adli(Howard Hughes Medical Institute), Todd Hricik(Memorial Sloan Kettering Cancer Center), Fan Liu(Memorial Sloan Kettering Cancer Center), Lindsay Saunders(Memorial Sloan Kettering Cancer Center), Ann Mullally(Brigham and Women's Hospital), Omar Abdel‐Wahab(Memorial Sloan Kettering Cancer Center), Laura Leung(Memorial Sloan Kettering Cancer Center), Abby Weinstein(Memorial Sloan Kettering Cancer Center), Sachie Marubayashi(Memorial Sloan Kettering Cancer Center), Aviva Goel(Memorial Sloan Kettering Cancer Center), Mithat Gönen(Memorial Sloan Kettering Cancer Center), Zeev Estrov(The University of Texas MD Anderson Cancer Center), Benjamin L. Ebert(Harvard University), Gabriela Chiosis(Memorial Sloan Kettering Cancer Center), Stephen D. Nimer(Memorial Sloan Kettering Cancer Center), B Bernstein(Broad Institute), Srđan Verstovšek(The University of Texas MD Anderson Cancer Center), Ross L. Levine(Memorial Sloan Kettering Cancer Center)
Nature
July 20, 2012
10.1038/nature11303
Cited by 366Open Access
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